Preliminary reports reveal conolidine may perhaps inhibit certain ion channels, decreasing neuronal excitability and limiting agony signals. This mechanism is particularly related in neuropathic soreness, where irregular signaling causes persistent pain. In addition, conolidine seems to impact G protein-coupled receptor (GPCR) pathways integral to agony notion. Computational docking s... https://ashleyl740gev2.wikiap.com/user